Thanks
to years of cross platform cooperation between researchers, governing bodies,
financial institutions, and substantial community participation, two general
forms of vaccines have been implemented to effect a considerable interruption
in the transmission of polio in the Americas. Polio-free zones are currently
present or emerging in the Americas, northern, southern, and eastern Africa,
the Arabian Peninsula, Western and Central Europe, and the Western Pacific.
Yet as encouraging as these facts may seem, several threats still exist
that can easily bring about another polio epidemic.
1.
Reversion
The
oral polio vaccine developed by Sabin uses an attenutated poliovirus developed
via serial passage selection through monkey kidney cells in the US and
human fibroblasts elsewhere. The attenuated poliovirus, however,
can still revert to the virulent form either via back mutation to the virulent
wild type form or recombination with other types to re-attain the necessary
gene sequence to produce the internal ribosome entry site (IRES) necessary
for infection and manifestation into disease.
2.
Vaccinees as healthy carriers
A vaccinated
person, whose attenuated poliovirus has reverted, will appear healthy for
nearly 30 days due to the incubation period which can last for as long
as 35 days. During that period, the person can spread the reverted
poliovirus either fecally or orally without exhibiting any symptoms of
polio and thus transforming him into a "healthy carrier."
3.
Potential Hot-zonesDespite
substantial progress, paralytic polio remains highly endemic throughout
the India subcontinent and continues to thrive in sub-Saharan Africa, Asia,
and several republics of the former Soviet Union.Poor
sanitation, crowding, lack of routine vaccination, and pockets of unvaccinated
children contribute to the persistence of polio in these areas. With
the high rate of intercontinental travel, these reservoirs of polio still
present a high risk to the polio-free western world.
1.PCR
technique for identifying wild-type poliovirus in clinical specimens and
environmental samples.
2.Nucleic acid hybridization to specifically identify all wild-type polioviruses.
3.Restriction fragments length polymorphism (RFLP).
4.Use of mouse cell lines expressing the human poliovirus receptor.
5.Use
of Sabin-specific monoclonal antibodies for rapid identification of Sabin-related
poliovirus by neutralisation.
6.Development
of a standard test for detection of poliovirus-specific IgM antibodies
to evaluate transgenic mice as an animal model for neurovirulence test
of oral poliomyelitis vaccine.
http://www.who.org/vaccines-diseases/research/virus1.htm